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This observation is intriguing for two other causes. 1st, it indicates that cells may take up MIF by ENDOCYTOSIS. supplied the plentiful intracellular expression of MIF, it'd be requested what advantage would the cell gain by making use of endocytosed MIF in lieu of intracellular MIF. Endocytosis of MIF may well come about either within a receptor-
Also, insulin from MIF-deficient mice experienced a unique conformation or posttranslational modification compared to insulin from wildtype mice. MIF in fact stimulated insulin hexamer formation in mobile-no cost devices [one hundred]. Hence, evidently MIF can work as an adaptor protein for insulin oligomerization. Insulin was also located to co-im